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DESCRIBE <http://purl.uniprot.org/SHA-384/FB46B922E97A71C1C365C0C0343FAFA89E551C541FC9585BFCBBCC14E2917DDB6139643477D09223816341D5BD3ABEDD>
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http://purl.uniprot.org/SHA-384/FB46B922E97A71C1C365C0C0343FAFA89E551C541FC9585BFCBBCC14E2917DDB6139643477D09223816341D5BD3ABEDD
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/FB46B922E97A71C1C365C0C0343FAFA89E551C541FC9585BFCBBCC14E2917DDB6139643477D09223816341D5BD3ABEDD
http://www.w3.org/2000/01/rdf-schema#comment
"Results suggest that SRC-2 coordinates cardiomyocyte secretion of VEGF downstream of the two major angiogenic stimuli occurring during pressure overload bridging both hypertrophic and hypoxia-stimulated paracrine signaling."
xsd:string
http://purl.uniprot.org/uniprot/#_83ED308BE40E9F4043A7206727FED80D8EE31E14E4999542B9DBEE07A450146FEBEA34EB3705B9B75C1D649EF33C83B7
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/FB46B922E97A71C1C365C0C0343FAFA89E551C541FC9585BFCBBCC14E2917DDB6139643477D09223816341D5BD3ABEDD
http://purl.uniprot.org/uniprot/Q8CAG7
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/FB46B922E97A71C1C365C0C0343FAFA89E551C541FC9585BFCBBCC14E2917DDB6139643477D09223816341D5BD3ABEDD
http://purl.uniprot.org/uniprot/#_Q8CAG7-mappedCitation-29127200
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/FB46B922E97A71C1C365C0C0343FAFA89E551C541FC9585BFCBBCC14E2917DDB6139643477D09223816341D5BD3ABEDD