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DESCRIBE <http://purl.uniprot.org/SHA-384/FC2BA12F6A875F67EAAB169C8E81E777808E34CEDF21CC9F0103F7C0EEDFF13CC631DBC79E1CF62DC6574BF01D9D07E6>
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http://purl.uniprot.org/SHA-384/FC2BA12F6A875F67EAAB169C8E81E777808E34CEDF21CC9F0103F7C0EEDFF13CC631DBC79E1CF62DC6574BF01D9D07E6
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/FC2BA12F6A875F67EAAB169C8E81E777808E34CEDF21CC9F0103F7C0EEDFF13CC631DBC79E1CF62DC6574BF01D9D07E6
http://www.w3.org/2000/01/rdf-schema#comment
"SAA stimulates CCL2 production via FPRL1 and thus contributes to atherosclerosis."
xsd:string
http://purl.uniprot.org/uniprot/#_D717791A759CE656DBEA522DE9109C613D30394452D42AB3828565CFB3D13B9BA11D3F008A02BF0B4A2A285A3229A767
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/FC2BA12F6A875F67EAAB169C8E81E777808E34CEDF21CC9F0103F7C0EEDFF13CC631DBC79E1CF62DC6574BF01D9D07E6
http://purl.uniprot.org/uniprot/P21462
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/FC2BA12F6A875F67EAAB169C8E81E777808E34CEDF21CC9F0103F7C0EEDFF13CC631DBC79E1CF62DC6574BF01D9D07E6
http://purl.uniprot.org/uniprot/#_P21462-mappedCitation-19167353
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/FC2BA12F6A875F67EAAB169C8E81E777808E34CEDF21CC9F0103F7C0EEDFF13CC631DBC79E1CF62DC6574BF01D9D07E6