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DESCRIBE <http://purl.uniprot.org/SHA-384/FDC74E8E23C46528326CF4D2E6A05A0BCF805214AAF1CD1C7F43D8355F90AE90227CE4C3797C5C1118E6E4D3C6C6EF54>
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http://purl.uniprot.org/SHA-384/FDC74E8E23C46528326CF4D2E6A05A0BCF805214AAF1CD1C7F43D8355F90AE90227CE4C3797C5C1118E6E4D3C6C6EF54
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/FDC74E8E23C46528326CF4D2E6A05A0BCF805214AAF1CD1C7F43D8355F90AE90227CE4C3797C5C1118E6E4D3C6C6EF54
http://www.w3.org/2000/01/rdf-schema#comment
"The main pathway by which Fas signaling regulates the levels of Bim expression in human T-cell blasts is the death-domain- and caspase-independent generation of discrete levels of H2O2 which results in the net increase of Foxo3a levels."
xsd:string
http://purl.uniprot.org/uniprot/#_6B3904F0853B8D3C27E8A65388AC41FB9C60957B446DD7CA52ED4823A1A588683CB76611A976ECA2E618FD68C9F3E148
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/FDC74E8E23C46528326CF4D2E6A05A0BCF805214AAF1CD1C7F43D8355F90AE90227CE4C3797C5C1118E6E4D3C6C6EF54
http://purl.uniprot.org/uniprot/O43521
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/FDC74E8E23C46528326CF4D2E6A05A0BCF805214AAF1CD1C7F43D8355F90AE90227CE4C3797C5C1118E6E4D3C6C6EF54
http://purl.uniprot.org/uniprot/#_O43521-mappedCitation-17062728
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/FDC74E8E23C46528326CF4D2E6A05A0BCF805214AAF1CD1C7F43D8355F90AE90227CE4C3797C5C1118E6E4D3C6C6EF54