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DESCRIBE <http://purl.uniprot.org/SHA-384/FFE5DB5D7C0F9568FF917A21A8346A75B3EE096A7060CE0B41FD07EBE126A54CE52758496F840420BF421F9616B9A2CF>
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http://purl.uniprot.org/SHA-384/FFE5DB5D7C0F9568FF917A21A8346A75B3EE096A7060CE0B41FD07EBE126A54CE52758496F840420BF421F9616B9A2CF
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/FFE5DB5D7C0F9568FF917A21A8346A75B3EE096A7060CE0B41FD07EBE126A54CE52758496F840420BF421F9616B9A2CF
http://www.w3.org/2000/01/rdf-schema#comment
"This report describes the preservation of heterozygous c.402C>G FOXL2 mutation in recurrent aGCTs. This finding adds further credence to the concept that the c.402C>G FOXL2 mutation is oncogenic and integral to this disease."
xsd:string
http://purl.uniprot.org/uniprot/#_8B5C5ADD9564BF6765F6A79DE3B36691C3D0110C616576BBEEE02970D360868AC0FB977189CCE7162B88F7AB2ABDA9ED
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/FFE5DB5D7C0F9568FF917A21A8346A75B3EE096A7060CE0B41FD07EBE126A54CE52758496F840420BF421F9616B9A2CF
http://purl.uniprot.org/uniprot/Q53ZD3
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/FFE5DB5D7C0F9568FF917A21A8346A75B3EE096A7060CE0B41FD07EBE126A54CE52758496F840420BF421F9616B9A2CF
http://purl.uniprot.org/uniprot/#_Q53ZD3-mappedCitation-28594898
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/FFE5DB5D7C0F9568FF917A21A8346A75B3EE096A7060CE0B41FD07EBE126A54CE52758496F840420BF421F9616B9A2CF