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http://purl.uniprot.org/SHA-384/FFE5EAC69F36E92096B7ADAA1B8ACDD1A4F6D8CC222CABA6FEF6007645034E1A9543A4DC8DE90F3C57E158AC83230574http://www.w3.org/1999/02/22-rdf-syntax-ns#typehttp://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/FFE5EAC69F36E92096B7ADAA1B8ACDD1A4F6D8CC222CABA6FEF6007645034E1A9543A4DC8DE90F3C57E158AC83230574http://www.w3.org/2000/01/rdf-schema#comment"MCM10 is highly expressed in lung cancer (LC) clinical specimens and significantly associated with recurrence pathological stage and worse overall survival. MCM10 knockdown in A549 and H661 cell lines significantly suppressed cell viability clone formation and induced G1 phase arrest by regulating the expression of CCND1. Results indicated a combined effect of MCM10CCND1 in predicting the prognosis of LC patients."xsd:string
http://purl.uniprot.org/uniprot/#_566677CF908160037F68A9237C1C7C000016523FBF973933E45284D4673D3A1D6EAE479FB751269D473500FE06288111http://www.w3.org/1999/02/22-rdf-syntax-ns#subjecthttp://purl.uniprot.org/SHA-384/FFE5EAC69F36E92096B7ADAA1B8ACDD1A4F6D8CC222CABA6FEF6007645034E1A9543A4DC8DE90F3C57E158AC83230574
http://purl.uniprot.org/uniprot/Q5T670http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/FFE5EAC69F36E92096B7ADAA1B8ACDD1A4F6D8CC222CABA6FEF6007645034E1A9543A4DC8DE90F3C57E158AC83230574
http://purl.uniprot.org/uniprot/#_Q5T670-mappedCitation-31638210http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/FFE5EAC69F36E92096B7ADAA1B8ACDD1A4F6D8CC222CABA6FEF6007645034E1A9543A4DC8DE90F3C57E158AC83230574