http://purl.uniprot.org/citations/10101682 | http://www.w3.org/1999/02/22-rdf-syntax-ns#type | http://purl.uniprot.org/core/Journal_Citation |
http://purl.uniprot.org/citations/10101682 | http://www.w3.org/2000/01/rdf-schema#comment | "Nineteen mAb directed against murine fusion regulatory protein-1 (mFRP-1)/4F2/CD98 were isolated and their biological properties were analysed. Intriguingly, mFRP-1 was found to be an alloantigen, namely, FRP-1.1 (DBA/2 and CBA mice type) and FRP-1.2 (BALB/c, C57BL/6 and C3H/He mice type). The nucleotide sequences of FRP-1.1 and FRP-1.2 were determined, demonstrating that amino acid change at 129 (P<-->R) is related to the alloantigenicity. mFRP-1 is expressed on thymocytes, on spleen cells, on peripheral lymphocytes and on blood monocytes, suggesting that the physiological role in vivo of murine FRP-1 is different from that of human FRP-1. The biological activities of antimFRP-1 mAbs showed by the present study are: (i) enhancement of Newcastle disease virus-induced cell fusion; (ii) suppression of HIVgp160-mediated cell fusion; and (iii) induction of aggregation and multinucleated giant cells of monocytes/macrophages."xsd:string |
http://purl.uniprot.org/citations/10101682 | http://purl.org/dc/terms/identifier | "doi:10.1046/j.1440-1711.1999.00792.x"xsd:string |
http://purl.uniprot.org/citations/10101682 | http://purl.uniprot.org/core/author | "Ito Y."xsd:string |
http://purl.uniprot.org/citations/10101682 | http://purl.uniprot.org/core/author | "Tajima M."xsd:string |
http://purl.uniprot.org/citations/10101682 | http://purl.uniprot.org/core/author | "Kawano M."xsd:string |
http://purl.uniprot.org/citations/10101682 | http://purl.uniprot.org/core/author | "Komada H."xsd:string |
http://purl.uniprot.org/citations/10101682 | http://purl.uniprot.org/core/author | "Kozuka Y."xsd:string |
http://purl.uniprot.org/citations/10101682 | http://purl.uniprot.org/core/author | "Tsumura H."xsd:string |
http://purl.uniprot.org/citations/10101682 | http://purl.uniprot.org/core/author | "Tsurudome M."xsd:string |
http://purl.uniprot.org/citations/10101682 | http://purl.uniprot.org/core/author | "Nishio M."xsd:string |
http://purl.uniprot.org/citations/10101682 | http://purl.uniprot.org/core/author | "Kusagawa S."xsd:string |
http://purl.uniprot.org/citations/10101682 | http://purl.uniprot.org/core/author | "Shimura K."xsd:string |
http://purl.uniprot.org/citations/10101682 | http://purl.uniprot.org/core/author | "Yoshimura S."xsd:string |
http://purl.uniprot.org/citations/10101682 | http://purl.uniprot.org/core/author | "Kusaura T."xsd:string |
http://purl.uniprot.org/citations/10101682 | http://purl.uniprot.org/core/date | "1999"xsd:gYear |
http://purl.uniprot.org/citations/10101682 | http://purl.uniprot.org/core/name | "Immunol Cell Biol"xsd:string |
http://purl.uniprot.org/citations/10101682 | http://purl.uniprot.org/core/pages | "19-27"xsd:string |
http://purl.uniprot.org/citations/10101682 | http://purl.uniprot.org/core/title | "Isolation and characterization of monoclonal antibodies directed against murine FRP-1/CD98/4F2 heavy chain: murine FRP-1 is an alloantigen and amino acid change at 129 (P<-->R) is related to the alloantigenicity."xsd:string |
http://purl.uniprot.org/citations/10101682 | http://purl.uniprot.org/core/volume | "77"xsd:string |
http://purl.uniprot.org/citations/10101682 | http://www.w3.org/2004/02/skos/core#exactMatch | http://purl.uniprot.org/pubmed/10101682 |
http://purl.uniprot.org/citations/10101682 | http://xmlns.com/foaf/0.1/primaryTopicOf | https://pubmed.ncbi.nlm.nih.gov/10101682 |
http://purl.uniprot.org/uniprot/#_A0A0U1RP32-mappedCitation-10101682 | http://www.w3.org/1999/02/22-rdf-syntax-ns#object | http://purl.uniprot.org/citations/10101682 |
http://purl.uniprot.org/uniprot/#_A0A0U1RP98-mappedCitation-10101682 | http://www.w3.org/1999/02/22-rdf-syntax-ns#object | http://purl.uniprot.org/citations/10101682 |
http://purl.uniprot.org/uniprot/#_A0A0U1RPG5-mappedCitation-10101682 | http://www.w3.org/1999/02/22-rdf-syntax-ns#object | http://purl.uniprot.org/citations/10101682 |