| http://purl.uniprot.org/citations/15371357 | http://www.w3.org/1999/02/22-rdf-syntax-ns#type | http://purl.uniprot.org/core/Journal_Citation |
| http://purl.uniprot.org/citations/15371357 | http://www.w3.org/1999/02/22-rdf-syntax-ns#type | http://purl.uniprot.org/core/Journal_Citation |
| http://purl.uniprot.org/citations/15371357 | http://www.w3.org/2000/01/rdf-schema#comment | "During Caenorhabditis elegans development, the HSN neurons and the right Q neuroblast and its descendants undergo long-range anteriorly directed migrations. Both of these migrations require EGL-20, a C. elegans Wnt homolog. Through a canonical Wnt signaling pathway, EGL-20/Wnt transcriptionally activates the Hox gene mab-5 in the left Q neuroblast and its descendants, causing the cells to migrate posteriorly. In this report, we show that CAM-1, a Ror receptor tyrosine kinase (RTK) family member, inhibits EGL-20 signaling. Excess EGL-20, like loss of cam-1, caused the HSNs to migrate too far anteriorly. Excess CAM-1, like loss of egl-20, shifted the final positions of the HSNs posteriorly and caused the left Q neuroblast descendants to migrate anteriorly. The reversal in the migration of the left Q neuroblast and its descendants resulted from a failure to express mab-5, an egl-20 mutant phenotype. Our data suggest that CAM-1 negatively regulates EGL-20."xsd:string |
| http://purl.uniprot.org/citations/15371357 | http://purl.org/dc/terms/identifier | "doi:10.1534/genetics.104.031781"xsd:string |
| http://purl.uniprot.org/citations/15371357 | http://purl.org/dc/terms/identifier | "doi:10.1534/genetics.104.031781"xsd:string |
| http://purl.uniprot.org/citations/15371357 | http://purl.uniprot.org/core/author | "Kim C."xsd:string |
| http://purl.uniprot.org/citations/15371357 | http://purl.uniprot.org/core/author | "Kim C."xsd:string |
| http://purl.uniprot.org/citations/15371357 | http://purl.uniprot.org/core/author | "Garriga G."xsd:string |
| http://purl.uniprot.org/citations/15371357 | http://purl.uniprot.org/core/author | "Garriga G."xsd:string |
| http://purl.uniprot.org/citations/15371357 | http://purl.uniprot.org/core/author | "Forrester W.C."xsd:string |
| http://purl.uniprot.org/citations/15371357 | http://purl.uniprot.org/core/author | "Forrester W.C."xsd:string |
| http://purl.uniprot.org/citations/15371357 | http://purl.uniprot.org/core/date | "2004"xsd:gYear |
| http://purl.uniprot.org/citations/15371357 | http://purl.uniprot.org/core/date | "2004"xsd:gYear |
| http://purl.uniprot.org/citations/15371357 | http://purl.uniprot.org/core/name | "Genetics"xsd:string |
| http://purl.uniprot.org/citations/15371357 | http://purl.uniprot.org/core/name | "Genetics"xsd:string |
| http://purl.uniprot.org/citations/15371357 | http://purl.uniprot.org/core/pages | "1951-1962"xsd:string |
| http://purl.uniprot.org/citations/15371357 | http://purl.uniprot.org/core/pages | "1951-1962"xsd:string |
| http://purl.uniprot.org/citations/15371357 | http://purl.uniprot.org/core/title | "The Caenorhabditis elegans Ror RTK CAM-1 inhibits EGL-20/Wnt signaling in cell migration."xsd:string |
| http://purl.uniprot.org/citations/15371357 | http://purl.uniprot.org/core/title | "The Caenorhabditis elegans Ror RTK CAM-1 inhibits EGL-20/Wnt signaling in cell migration."xsd:string |
| http://purl.uniprot.org/citations/15371357 | http://purl.uniprot.org/core/volume | "168"xsd:string |
| http://purl.uniprot.org/citations/15371357 | http://purl.uniprot.org/core/volume | "168"xsd:string |
| http://purl.uniprot.org/citations/15371357 | http://www.w3.org/2004/02/skos/core#exactMatch | http://purl.uniprot.org/pubmed/15371357 |
| http://purl.uniprot.org/citations/15371357 | http://www.w3.org/2004/02/skos/core#exactMatch | http://purl.uniprot.org/pubmed/15371357 |
| http://purl.uniprot.org/citations/15371357 | http://xmlns.com/foaf/0.1/primaryTopicOf | https://pubmed.ncbi.nlm.nih.gov/15371357 |
| http://purl.uniprot.org/citations/15371357 | http://xmlns.com/foaf/0.1/primaryTopicOf | https://pubmed.ncbi.nlm.nih.gov/15371357 |