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http://purl.uniprot.org/citations/16139274http://www.w3.org/1999/02/22-rdf-syntax-ns#typehttp://purl.uniprot.org/core/Journal_Citation
http://purl.uniprot.org/citations/16139274http://www.w3.org/2000/01/rdf-schema#comment"Cannabinoids have been shown to inhibit the growth of a broad spectrum of tumour cells. However, the molecular mechanisms involved in that effect have not been completely elucidated. Here, we investigated the possible involvement of mitogen-activated protein kinases (MAPKs) in CB2 receptor-induced apoptosis of human leukaemia cells. Results show that stimulation of the CB2 receptor leads to p38 MAPK activation and that inhibition of this kinase attenuates CB2 receptor-induced caspase activation and apoptosis. These findings support a role for p38 MAPK in CB2 receptor-induced apoptosis of human leukaemia cells."xsd:string
http://purl.uniprot.org/citations/16139274http://purl.org/dc/terms/identifier"doi:10.1016/j.febslet.2005.08.021"xsd:string
http://purl.uniprot.org/citations/16139274http://purl.uniprot.org/core/author"Carracedo A."xsd:string
http://purl.uniprot.org/citations/16139274http://purl.uniprot.org/core/author"Velasco G."xsd:string
http://purl.uniprot.org/citations/16139274http://purl.uniprot.org/core/author"Guzman M."xsd:string
http://purl.uniprot.org/citations/16139274http://purl.uniprot.org/core/author"Herrera B."xsd:string
http://purl.uniprot.org/citations/16139274http://purl.uniprot.org/core/author"Diez-Zaera M."xsd:string
http://purl.uniprot.org/citations/16139274http://purl.uniprot.org/core/date"2005"xsd:gYear
http://purl.uniprot.org/citations/16139274http://purl.uniprot.org/core/name"FEBS Lett"xsd:string
http://purl.uniprot.org/citations/16139274http://purl.uniprot.org/core/pages"5084-5088"xsd:string
http://purl.uniprot.org/citations/16139274http://purl.uniprot.org/core/title"p38 MAPK is involved in CB2 receptor-induced apoptosis of human leukaemia cells."xsd:string
http://purl.uniprot.org/citations/16139274http://purl.uniprot.org/core/volume"579"xsd:string
http://purl.uniprot.org/citations/16139274http://www.w3.org/2004/02/skos/core#exactMatchhttp://purl.uniprot.org/pubmed/16139274
http://purl.uniprot.org/citations/16139274http://xmlns.com/foaf/0.1/primaryTopicOfhttps://pubmed.ncbi.nlm.nih.gov/16139274
http://purl.uniprot.org/uniprot/#_A0A510GDE6-mappedCitation-16139274http://www.w3.org/1999/02/22-rdf-syntax-ns#objecthttp://purl.uniprot.org/citations/16139274
http://purl.uniprot.org/uniprot/#_B4E0K5-mappedCitation-16139274http://www.w3.org/1999/02/22-rdf-syntax-ns#objecthttp://purl.uniprot.org/citations/16139274
http://purl.uniprot.org/uniprot/#_B5TY33-mappedCitation-16139274http://www.w3.org/1999/02/22-rdf-syntax-ns#objecthttp://purl.uniprot.org/citations/16139274
http://purl.uniprot.org/uniprot/#_Q15454-mappedCitation-16139274http://www.w3.org/1999/02/22-rdf-syntax-ns#objecthttp://purl.uniprot.org/citations/16139274
http://purl.uniprot.org/uniprot/#_L7RSM2-mappedCitation-16139274http://www.w3.org/1999/02/22-rdf-syntax-ns#objecthttp://purl.uniprot.org/citations/16139274
http://purl.uniprot.org/uniprot/#_Q16539-mappedCitation-16139274http://www.w3.org/1999/02/22-rdf-syntax-ns#objecthttp://purl.uniprot.org/citations/16139274
http://purl.uniprot.org/uniprot/#_P34972-mappedCitation-16139274http://www.w3.org/1999/02/22-rdf-syntax-ns#objecthttp://purl.uniprot.org/citations/16139274
http://purl.uniprot.org/uniprot/A0A510GDE6http://purl.uniprot.org/core/mappedCitationhttp://purl.uniprot.org/citations/16139274
http://purl.uniprot.org/uniprot/P34972http://purl.uniprot.org/core/mappedCitationhttp://purl.uniprot.org/citations/16139274
http://purl.uniprot.org/uniprot/L7RSM2http://purl.uniprot.org/core/mappedCitationhttp://purl.uniprot.org/citations/16139274