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http://purl.uniprot.org/citations/16964398http://www.w3.org/1999/02/22-rdf-syntax-ns#typehttp://purl.uniprot.org/core/Journal_Citation
http://purl.uniprot.org/citations/16964398http://www.w3.org/2000/01/rdf-schema#comment"EG-1 is significantly elevated in breast, colorectal, and prostate cancers. Overexpression of EG-1 stimulates cellular proliferation, and targeted inhibition blocks mouse xenograft tumor growth. To further clarify the function of EG-1, we investigated its role in c-Src activation. We observed that EG-1 overexpression results in activation of c-Src, but found no evidence that EG-1 is a direct Src substrate. EG-1 also binds to other members of the Src family. Furthermore, EG-1 shows interaction with multiple other SH3- and WW-containing molecules involved in various signaling pathways. These observations suggest that EG-1 may be involved in signaling pathways including c-Src activation."xsd:string
http://purl.uniprot.org/citations/16964398http://purl.uniprot.org/core/author"Lu M."xsd:string
http://purl.uniprot.org/citations/16964398http://purl.uniprot.org/core/author"Zhang L."xsd:string
http://purl.uniprot.org/citations/16964398http://purl.uniprot.org/core/author"Norris A.J."xsd:string
http://purl.uniprot.org/citations/16964398http://purl.uniprot.org/core/author"Brooks M.N."xsd:string
http://purl.uniprot.org/citations/16964398http://purl.uniprot.org/core/author"Sartippour M.R."xsd:string
http://purl.uniprot.org/citations/16964398http://purl.uniprot.org/core/date"2006"xsd:gYear
http://purl.uniprot.org/citations/16964398http://purl.uniprot.org/core/name"Int J Oncol"xsd:string
http://purl.uniprot.org/citations/16964398http://purl.uniprot.org/core/pages"1013-1018"xsd:string
http://purl.uniprot.org/citations/16964398http://purl.uniprot.org/core/title"EG-1 interacts with c-Src and activates its signaling pathway."xsd:string
http://purl.uniprot.org/citations/16964398http://purl.uniprot.org/core/volume"29"xsd:string
http://purl.uniprot.org/citations/16964398http://www.w3.org/2004/02/skos/core#exactMatchhttp://purl.uniprot.org/pubmed/16964398
http://purl.uniprot.org/citations/16964398http://xmlns.com/foaf/0.1/primaryTopicOfhttps://pubmed.ncbi.nlm.nih.gov/16964398
http://purl.uniprot.org/uniprot/#_A8K3B6-mappedCitation-16964398http://www.w3.org/1999/02/22-rdf-syntax-ns#objecthttp://purl.uniprot.org/citations/16964398
http://purl.uniprot.org/uniprot/#_B2R6Q4-mappedCitation-16964398http://www.w3.org/1999/02/22-rdf-syntax-ns#objecthttp://purl.uniprot.org/citations/16964398
http://purl.uniprot.org/uniprot/#_B4DMZ0-mappedCitation-16964398http://www.w3.org/1999/02/22-rdf-syntax-ns#objecthttp://purl.uniprot.org/citations/16964398
http://purl.uniprot.org/uniprot/#_B4DP99-mappedCitation-16964398http://www.w3.org/1999/02/22-rdf-syntax-ns#objecthttp://purl.uniprot.org/citations/16964398
http://purl.uniprot.org/uniprot/#_B5BU52-mappedCitation-16964398http://www.w3.org/1999/02/22-rdf-syntax-ns#objecthttp://purl.uniprot.org/citations/16964398
http://purl.uniprot.org/uniprot/#_O15259-mappedCitation-16964398http://www.w3.org/1999/02/22-rdf-syntax-ns#objecthttp://purl.uniprot.org/citations/16964398
http://purl.uniprot.org/uniprot/#_P42680-mappedCitation-16964398http://www.w3.org/1999/02/22-rdf-syntax-ns#objecthttp://purl.uniprot.org/citations/16964398
http://purl.uniprot.org/uniprot/#_P42684-mappedCitation-16964398http://www.w3.org/1999/02/22-rdf-syntax-ns#objecthttp://purl.uniprot.org/citations/16964398
http://purl.uniprot.org/uniprot/#_P06241-mappedCitation-16964398http://www.w3.org/1999/02/22-rdf-syntax-ns#objecthttp://purl.uniprot.org/citations/16964398
http://purl.uniprot.org/uniprot/#_P41240-mappedCitation-16964398http://www.w3.org/1999/02/22-rdf-syntax-ns#objecthttp://purl.uniprot.org/citations/16964398
http://purl.uniprot.org/uniprot/#_P27986-mappedCitation-16964398http://www.w3.org/1999/02/22-rdf-syntax-ns#objecthttp://purl.uniprot.org/citations/16964398