http://purl.uniprot.org/citations/24357608 | http://www.w3.org/1999/02/22-rdf-syntax-ns#type | http://purl.uniprot.org/core/Journal_Citation |
http://purl.uniprot.org/citations/24357608 | http://www.w3.org/1999/02/22-rdf-syntax-ns#type | http://purl.uniprot.org/core/Journal_Citation |
http://purl.uniprot.org/citations/24357608 | http://www.w3.org/2000/01/rdf-schema#comment | "Recognition of microbial challenges leads to enhanced immunity at both the local and systemic levels. In Arabidopsis, EFR and PEPR1/PEPR2 act as the receptor for the bacterial elongation factor EF-Tu (elf18 epitope) and for the endogenous PROPEP-derived Pep epitopes, respectively. The PEPR pathway has been described to mediate defence signalling following microbial recognition. Here we show that PROPEP2/PROPEP3 induction upon pathogen challenges is robust against jasmonate, salicylate, or ethylene dysfunction. Comparative transcriptome profiling between Pep2- and elf18-treated plants points to co-activation of otherwise antagonistic jasmonate- and salicylate-mediated immune branches as a key output of PEPR signalling. Accordingly, as well as basal defences against hemibiotrophic pathogens, systemic immunity is reduced in pepr1 pepr2 plants. Remarkably, PROPEP2/PROPEP3 induction is essentially restricted to the pathogen challenge sites during pathogen-induced systemic immunity. Localized Pep application activates genetically separable jasmonate and salicylate branches in systemic leaves without significant PROPEP2/PROPEP3 induction. Our results suggest that local PEPR activation provides a critical step in connecting local to systemic immunity by reinforcing separate defence signalling pathways."xsd:string |
http://purl.uniprot.org/citations/24357608 | http://purl.org/dc/terms/identifier | "doi:10.1002/embj.201284303"xsd:string |
http://purl.uniprot.org/citations/24357608 | http://purl.org/dc/terms/identifier | "doi:10.1002/embj.201284303"xsd:string |
http://purl.uniprot.org/citations/24357608 | http://purl.uniprot.org/core/author | "Lu X."xsd:string |
http://purl.uniprot.org/citations/24357608 | http://purl.uniprot.org/core/author | "Lu X."xsd:string |
http://purl.uniprot.org/citations/24357608 | http://purl.uniprot.org/core/author | "Hiruma K."xsd:string |
http://purl.uniprot.org/citations/24357608 | http://purl.uniprot.org/core/author | "Hiruma K."xsd:string |
http://purl.uniprot.org/citations/24357608 | http://purl.uniprot.org/core/author | "Takano Y."xsd:string |
http://purl.uniprot.org/citations/24357608 | http://purl.uniprot.org/core/author | "Takano Y."xsd:string |
http://purl.uniprot.org/citations/24357608 | http://purl.uniprot.org/core/author | "Yamada K."xsd:string |
http://purl.uniprot.org/citations/24357608 | http://purl.uniprot.org/core/author | "Yamada K."xsd:string |
http://purl.uniprot.org/citations/24357608 | http://purl.uniprot.org/core/author | "Tsuda K."xsd:string |
http://purl.uniprot.org/citations/24357608 | http://purl.uniprot.org/core/author | "Tsuda K."xsd:string |
http://purl.uniprot.org/citations/24357608 | http://purl.uniprot.org/core/author | "Ross A."xsd:string |
http://purl.uniprot.org/citations/24357608 | http://purl.uniprot.org/core/author | "Ross A."xsd:string |
http://purl.uniprot.org/citations/24357608 | http://purl.uniprot.org/core/author | "Saijo Y."xsd:string |
http://purl.uniprot.org/citations/24357608 | http://purl.uniprot.org/core/author | "Saijo Y."xsd:string |
http://purl.uniprot.org/citations/24357608 | http://purl.uniprot.org/core/author | "Yamashita-Yamada M."xsd:string |
http://purl.uniprot.org/citations/24357608 | http://purl.uniprot.org/core/author | "Yamashita-Yamada M."xsd:string |
http://purl.uniprot.org/citations/24357608 | http://purl.uniprot.org/core/date | "2014"xsd:gYear |
http://purl.uniprot.org/citations/24357608 | http://purl.uniprot.org/core/date | "2014"xsd:gYear |
http://purl.uniprot.org/citations/24357608 | http://purl.uniprot.org/core/name | "EMBO J."xsd:string |
http://purl.uniprot.org/citations/24357608 | http://purl.uniprot.org/core/name | "EMBO J."xsd:string |