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| http://purl.uniprot.org/citations/24509466 | http://www.w3.org/2000/01/rdf-schema#comment | "Germline mutations in the exonuclease (proofreading) domains of 2 DNA polymerases (POLE and POLD1) have been associated with a dominantly inherited, highly penetrant syndrome of oligo adenomatous polyposis and early-age-of-diagnosis colorectal cancer and endometrial cancer. The loss of proofreading capability causes multiple mutations throughout the genome and is manifest as microsatellite-stable, chromosomal unstable cancers. This is an important addition to the range of dominantly inherited syndromes of colorectal cancer predisposition."xsd:string |
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| http://purl.uniprot.org/citations/24509466 | http://purl.uniprot.org/core/author | "Church J.M."xsd:string |
| http://purl.uniprot.org/citations/24509466 | http://purl.uniprot.org/core/date | "2014"xsd:gYear |
| http://purl.uniprot.org/citations/24509466 | http://purl.uniprot.org/core/name | "Dis Colon Rectum"xsd:string |
| http://purl.uniprot.org/citations/24509466 | http://purl.uniprot.org/core/pages | "396-397"xsd:string |
| http://purl.uniprot.org/citations/24509466 | http://purl.uniprot.org/core/title | "Polymerase proofreading-associated polyposis: a new, dominantly inherited syndrome of hereditary colorectal cancer predisposition."xsd:string |
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