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| http://purl.uniprot.org/citations/25566191 | http://www.w3.org/2000/01/rdf-schema#comment | "Cholecystokinin (CCK) signaling appears well conserved over evolution. In Drosophila, the CCK-like sulfakinins (DSKs) regulate aspects of gut function, satiety and food ingestion, hyperactivity and aggression, as well as escape-related locomotion and synaptic plasticity during neuromuscular junction development. Activity in the DSK-producing neurons is regulated by octopamine. We discuss mechanisms behind CCK function in satiety, aggression, and locomotion in some detail and highlight similarities to mammalian CCK signaling."xsd:string |
| http://purl.uniprot.org/citations/25566191 | http://purl.org/dc/terms/identifier | "doi:10.3389/fendo.2014.00219"xsd:string |
| http://purl.uniprot.org/citations/25566191 | http://purl.uniprot.org/core/author | "Williams M.J."xsd:string |
| http://purl.uniprot.org/citations/25566191 | http://purl.uniprot.org/core/author | "Nassel D.R."xsd:string |
| http://purl.uniprot.org/citations/25566191 | http://purl.uniprot.org/core/date | "2014"xsd:gYear |
| http://purl.uniprot.org/citations/25566191 | http://purl.uniprot.org/core/name | "Front Endocrinol (Lausanne)"xsd:string |
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| http://purl.uniprot.org/citations/25566191 | http://purl.uniprot.org/core/title | "Cholecystokinin-Like Peptide (DSK) in Drosophila, Not Only for Satiety Signaling."xsd:string |
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