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http://purl.uniprot.org/citations/25955817http://www.w3.org/1999/02/22-rdf-syntax-ns#typehttp://purl.uniprot.org/core/Journal_Citation
http://purl.uniprot.org/citations/25955817http://www.w3.org/2000/01/rdf-schema#comment"Natriuretic peptide receptor A (NPR-A) is the receptor for atrial natriuretic peptide (ANP) and brain natriuretic peptide (BNP). NPR-A plays critical physiological and pathophysiological roles in several target cell and tissue system processes, such as cell growth, apoptosis, proliferation, and inflammation. Accumulating data demonstrate that NPR-A is involved in immune and inflammatory reactions and is a potential target in inflammation treatment. It is expressed in various cancer cells and is important for tumor growth. A recent study indicated that NPR-A signaling can regulate stem cell recruitment and angiogenesis. This signaling can serve as a model for studying the linkage between inflammation and tumorigenesis. In this review we highlight the mechanisms by which NPR-A affects signaling pathways involved in inflammation and cancer, and we discuss its potential as a novel target in inflammation, cancer, and cancer-related inflammation."xsd:string
http://purl.uniprot.org/citations/25955817http://purl.org/dc/terms/identifier"doi:10.1615/critreveukaryotgeneexpr.2015012447"xsd:string
http://purl.uniprot.org/citations/25955817http://purl.uniprot.org/core/author"Li J."xsd:string
http://purl.uniprot.org/citations/25955817http://purl.uniprot.org/core/author"Li M."xsd:string
http://purl.uniprot.org/citations/25955817http://purl.uniprot.org/core/author"Wang J."xsd:string
http://purl.uniprot.org/citations/25955817http://purl.uniprot.org/core/author"Zhang J."xsd:string
http://purl.uniprot.org/citations/25955817http://purl.uniprot.org/core/author"Yan Y."xsd:string
http://purl.uniprot.org/citations/25955817http://purl.uniprot.org/core/author"Yang Y."xsd:string
http://purl.uniprot.org/citations/25955817http://purl.uniprot.org/core/author"Qu J."xsd:string
http://purl.uniprot.org/citations/25955817http://purl.uniprot.org/core/date"2015"xsd:gYear
http://purl.uniprot.org/citations/25955817http://purl.uniprot.org/core/name"Crit Rev Eukaryot Gene Expr"xsd:string
http://purl.uniprot.org/citations/25955817http://purl.uniprot.org/core/pages"41-46"xsd:string
http://purl.uniprot.org/citations/25955817http://purl.uniprot.org/core/title"NPR-A: A Therapeutic Target in Inflammation and Cancer."xsd:string
http://purl.uniprot.org/citations/25955817http://purl.uniprot.org/core/volume"25"xsd:string
http://purl.uniprot.org/citations/25955817http://www.w3.org/2004/02/skos/core#exactMatchhttp://purl.uniprot.org/pubmed/25955817
http://purl.uniprot.org/citations/25955817http://xmlns.com/foaf/0.1/primaryTopicOfhttps://pubmed.ncbi.nlm.nih.gov/25955817
http://purl.uniprot.org/uniprot/#_A0A140VJE6-mappedCitation-25955817http://www.w3.org/1999/02/22-rdf-syntax-ns#objecthttp://purl.uniprot.org/citations/25955817
http://purl.uniprot.org/uniprot/#_B0ZBF1-mappedCitation-25955817http://www.w3.org/1999/02/22-rdf-syntax-ns#objecthttp://purl.uniprot.org/citations/25955817
http://purl.uniprot.org/uniprot/#_B7Z4Y7-mappedCitation-25955817http://www.w3.org/1999/02/22-rdf-syntax-ns#objecthttp://purl.uniprot.org/citations/25955817
http://purl.uniprot.org/uniprot/#_B4DU06-mappedCitation-25955817http://www.w3.org/1999/02/22-rdf-syntax-ns#objecthttp://purl.uniprot.org/citations/25955817
http://purl.uniprot.org/uniprot/#_P16066-mappedCitation-25955817http://www.w3.org/1999/02/22-rdf-syntax-ns#objecthttp://purl.uniprot.org/citations/25955817
http://purl.uniprot.org/uniprot/#_Q8IXF8-mappedCitation-25955817http://www.w3.org/1999/02/22-rdf-syntax-ns#objecthttp://purl.uniprot.org/citations/25955817
http://purl.uniprot.org/uniprot/B7Z4Y7http://purl.uniprot.org/core/mappedCitationhttp://purl.uniprot.org/citations/25955817
http://purl.uniprot.org/uniprot/B0ZBF1http://purl.uniprot.org/core/mappedCitationhttp://purl.uniprot.org/citations/25955817