| http://purl.uniprot.org/citations/26966923 | http://www.w3.org/1999/02/22-rdf-syntax-ns#type | http://purl.uniprot.org/core/Journal_Citation |
| http://purl.uniprot.org/citations/26966923 | http://www.w3.org/2000/01/rdf-schema#comment | "Wounding induces systemic potentials in Arabidopsis thaliana that can be abolished by concomitant suppression of the GLUTAMATE RECEPTOR-LIKE GLR3.3 and GLR3.6 genes. However, the roles of specific GLR channels to these potentials remain unclear. Here I applied the Electrical Penetration Graph (EPG) to study the contribution of three GLR channels to wound-induced, systemically propagated electrical potentials in Arabidopsis thaliana. In contrast to recordings made with conventional rigs for whole-plant electrophysiology, the EPG allows for the unambiguous distinction of the phloem-propagated action potential (AP) from the electrical activity outside of the phloem. The data reported here suggest that: (a) the transmission of wound-induced, phloem-propagated AP to neighbor leaves, requires expression of GLR3.3 or GLR3.6, whereas GLR3.5 prevents its transmission to non-neighbor leaves; (b) the generation of wound-induced electrical signals outside the phloem network depends on GLR3.6 expression; and (c) wound-induced systemic potentials initiated in the shoot are transmitted to the root in the adult plant, which suggests a role for these electrical signals in coordinating the plant defenses in the shoot and in the root. Here, I propose a model for wound-induced systemic electrical signals at the molecular, cellular and anatomical level. In this model, GLR3.3 and GLR3.6 function as on switches for the propagation of wound-induced potentials beyond the wounded leaf, while GLR3.5 functions as an off switch that prevents the propagation of wound-induced electrical potentials to distal, non-neighbor leaves."xsd:string |
| http://purl.uniprot.org/citations/26966923 | http://purl.org/dc/terms/identifier | "doi:10.1080/15592324.2016.1161879"xsd:string |
| http://purl.uniprot.org/citations/26966923 | http://purl.uniprot.org/core/author | "Salvador-Recatala V."xsd:string |
| http://purl.uniprot.org/citations/26966923 | http://purl.uniprot.org/core/date | "2016"xsd:gYear |
| http://purl.uniprot.org/citations/26966923 | http://purl.uniprot.org/core/name | "Plant Signal Behav"xsd:string |
| http://purl.uniprot.org/citations/26966923 | http://purl.uniprot.org/core/pages | "e1161879"xsd:string |
| http://purl.uniprot.org/citations/26966923 | http://purl.uniprot.org/core/title | "New roles for the GLUTAMATE RECEPTOR-LIKE 3.3, 3.5, and 3.6 genes as on/off switches of wound-induced systemic electrical signals."xsd:string |
| http://purl.uniprot.org/citations/26966923 | http://purl.uniprot.org/core/volume | "11"xsd:string |
| http://purl.uniprot.org/citations/26966923 | http://www.w3.org/2004/02/skos/core#exactMatch | http://purl.uniprot.org/pubmed/26966923 |
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| http://purl.uniprot.org/uniprot/#_A0A1P8AYR0-mappedCitation-26966923 | http://www.w3.org/1999/02/22-rdf-syntax-ns#object | http://purl.uniprot.org/citations/26966923 |
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| http://purl.uniprot.org/uniprot/#_A0A1I9LP23-mappedCitation-26966923 | http://www.w3.org/1999/02/22-rdf-syntax-ns#object | http://purl.uniprot.org/citations/26966923 |
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| http://purl.uniprot.org/uniprot/F4ITQ2 | http://purl.uniprot.org/core/mappedCitation | http://purl.uniprot.org/citations/26966923 |
| http://purl.uniprot.org/uniprot/F4ITQ0 | http://purl.uniprot.org/core/mappedCitation | http://purl.uniprot.org/citations/26966923 |