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http://purl.uniprot.org/citations/27208542http://www.w3.org/1999/02/22-rdf-syntax-ns#typehttp://purl.uniprot.org/core/Journal_Citation
http://purl.uniprot.org/citations/27208542http://www.w3.org/2000/01/rdf-schema#comment"Several phytohormones have been demonstrated to be involved in iron (Fe) homeostasis. We took advantage of a salicylic acid (SA) biosynthesis defective mutant phytoalexin deficient 4 (pad4: T-DNA Salk_089936) to explore the possible effects of endogenous SA on the morphological and physiological responses to Fe deprivation. The morphological and physiological analysis was carried out between Col-0 and the pad4 mutant. Under an Fe-deficiency treatment, Col-0 showed more severe leaf chlorosis and root growth inhibition compared with the pad4 mutant. The soluble Fe concentrations were significantly higher in pad4 than in Col-0 under the Fe-deficiency treatment. Fe deficiency significantly induced SA accumulation in Col-0 and the loss-of-function of PAD4 blocked this process. The requirement of endogenous SA accumulation for Fe-deficiency responses was confirmed using a series of SA biosynthetic mutants and transgenic lines. Furthermore, a comparative RNA sequencing analysis of the whole seedling transcriptomes between Col-0 and the pad4 mutant was also performed. Based on the transcriptome data, the expression levels of many auxin- and ethylene-response genes were altered in pad4 compared with Col-0. Fe deficiency increases SA contents which elevates auxin and ethylene signalling, thereby activating Fe translocation via the bHLH38/39-mediated transcriptional regulation of downstream Fe genes."xsd:string
http://purl.uniprot.org/citations/27208542http://purl.org/dc/terms/identifier"doi:10.1093/jxb/erw196"xsd:string
http://purl.uniprot.org/citations/27208542http://purl.uniprot.org/core/author"Guo H."xsd:string
http://purl.uniprot.org/citations/27208542http://purl.uniprot.org/core/author"Sun T."xsd:string
http://purl.uniprot.org/citations/27208542http://purl.uniprot.org/core/author"Shen C."xsd:string
http://purl.uniprot.org/citations/27208542http://purl.uniprot.org/core/author"Zhang L."xsd:string
http://purl.uniprot.org/citations/27208542http://purl.uniprot.org/core/author"Wang H."xsd:string
http://purl.uniprot.org/citations/27208542http://purl.uniprot.org/core/author"Yang Y."xsd:string
http://purl.uniprot.org/citations/27208542http://purl.uniprot.org/core/author"Liu K."xsd:string
http://purl.uniprot.org/citations/27208542http://purl.uniprot.org/core/date"2016"xsd:gYear
http://purl.uniprot.org/citations/27208542http://purl.uniprot.org/core/name"J Exp Bot"xsd:string
http://purl.uniprot.org/citations/27208542http://purl.uniprot.org/core/pages"4179-4193"xsd:string
http://purl.uniprot.org/citations/27208542http://purl.uniprot.org/core/title"Involvement of endogenous salicylic acid in iron-deficiency responses in Arabidopsis."xsd:string
http://purl.uniprot.org/citations/27208542http://purl.uniprot.org/core/volume"67"xsd:string
http://purl.uniprot.org/citations/27208542http://www.w3.org/2004/02/skos/core#exactMatchhttp://purl.uniprot.org/pubmed/27208542
http://purl.uniprot.org/citations/27208542http://xmlns.com/foaf/0.1/primaryTopicOfhttps://pubmed.ncbi.nlm.nih.gov/27208542
http://purl.uniprot.org/uniprot/#_Q9S745-mappedCitation-27208542http://www.w3.org/1999/02/22-rdf-syntax-ns#objecthttp://purl.uniprot.org/citations/27208542
http://purl.uniprot.org/uniprot/#_Q9M1K1-mappedCitation-27208542http://www.w3.org/1999/02/22-rdf-syntax-ns#objecthttp://purl.uniprot.org/citations/27208542
http://purl.uniprot.org/uniprot/Q9M1K1http://purl.uniprot.org/core/mappedCitationhttp://purl.uniprot.org/citations/27208542
http://purl.uniprot.org/uniprot/Q9S745http://purl.uniprot.org/core/mappedCitationhttp://purl.uniprot.org/citations/27208542