RDF/XMLNTriplesTurtleShow queryShare
SubjectPredicateObject
http://purl.uniprot.org/citations/29615557http://www.w3.org/1999/02/22-rdf-syntax-ns#typehttp://purl.uniprot.org/core/Journal_Citation
http://purl.uniprot.org/citations/29615557http://www.w3.org/2000/01/rdf-schema#comment"In mammals, the Wnt/β-catenin signal transduction pathway regulates intestinal stem cell maintenance and proliferation, whereas Wnt pathway hyperactivation, resulting primarily from the inactivation of the tumor suppressor Adenomatous polyposis coli (APC), triggers the development of the vast majority of colorectal cancers. The Drosophila adult gut has recently emerged as a powerful model to elucidate the mechanisms by which Wingless/Wnt signaling regulates intestinal development, homeostasis, regeneration, and tumorigenesis. Herein, we review recent insights on the roles of Wnt signaling in Drosophila intestinal physiology and pathology."xsd:string
http://purl.uniprot.org/citations/29615557http://purl.org/dc/terms/identifier"doi:10.3390/jdb6020008"xsd:string
http://purl.uniprot.org/citations/29615557http://purl.uniprot.org/core/author"Ahmed Y."xsd:string
http://purl.uniprot.org/citations/29615557http://purl.uniprot.org/core/author"Benchabane H."xsd:string
http://purl.uniprot.org/citations/29615557http://purl.uniprot.org/core/author"Tian A."xsd:string
http://purl.uniprot.org/citations/29615557http://purl.uniprot.org/core/date"2018"xsd:gYear
http://purl.uniprot.org/citations/29615557http://purl.uniprot.org/core/name"J Dev Biol"xsd:string
http://purl.uniprot.org/citations/29615557http://purl.uniprot.org/core/pages"E8"xsd:string
http://purl.uniprot.org/citations/29615557http://purl.uniprot.org/core/title"Wingless/Wnt Signaling in Intestinal Development, Homeostasis, Regeneration and Tumorigenesis: A Drosophila Perspective."xsd:string
http://purl.uniprot.org/citations/29615557http://purl.uniprot.org/core/volume"6"xsd:string
http://purl.uniprot.org/citations/29615557http://www.w3.org/2004/02/skos/core#exactMatchhttp://purl.uniprot.org/pubmed/29615557
http://purl.uniprot.org/citations/29615557http://xmlns.com/foaf/0.1/primaryTopicOfhttps://pubmed.ncbi.nlm.nih.gov/29615557
http://purl.uniprot.org/uniprot/#_A0A0B4KI19-mappedCitation-29615557http://www.w3.org/1999/02/22-rdf-syntax-ns#objecthttp://purl.uniprot.org/citations/29615557
http://purl.uniprot.org/uniprot/#_D2NUH8-mappedCitation-29615557http://www.w3.org/1999/02/22-rdf-syntax-ns#objecthttp://purl.uniprot.org/citations/29615557
http://purl.uniprot.org/uniprot/#_A0A0B4LFY4-mappedCitation-29615557http://www.w3.org/1999/02/22-rdf-syntax-ns#objecthttp://purl.uniprot.org/citations/29615557
http://purl.uniprot.org/uniprot/#_A0A126GV30-mappedCitation-29615557http://www.w3.org/1999/02/22-rdf-syntax-ns#objecthttp://purl.uniprot.org/citations/29615557
http://purl.uniprot.org/uniprot/#_A0A1Z1CN92-mappedCitation-29615557http://www.w3.org/1999/02/22-rdf-syntax-ns#objecthttp://purl.uniprot.org/citations/29615557
http://purl.uniprot.org/uniprot/#_M9NEL5-mappedCitation-29615557http://www.w3.org/1999/02/22-rdf-syntax-ns#objecthttp://purl.uniprot.org/citations/29615557
http://purl.uniprot.org/uniprot/#_M9PG44-mappedCitation-29615557http://www.w3.org/1999/02/22-rdf-syntax-ns#objecthttp://purl.uniprot.org/citations/29615557
http://purl.uniprot.org/uniprot/#_M9PGB3-mappedCitation-29615557http://www.w3.org/1999/02/22-rdf-syntax-ns#objecthttp://purl.uniprot.org/citations/29615557
http://purl.uniprot.org/uniprot/#_M9PGI0-mappedCitation-29615557http://www.w3.org/1999/02/22-rdf-syntax-ns#objecthttp://purl.uniprot.org/citations/29615557
http://purl.uniprot.org/uniprot/#_M9NE19-mappedCitation-29615557http://www.w3.org/1999/02/22-rdf-syntax-ns#objecthttp://purl.uniprot.org/citations/29615557
http://purl.uniprot.org/uniprot/#_P09615-mappedCitation-29615557http://www.w3.org/1999/02/22-rdf-syntax-ns#objecthttp://purl.uniprot.org/citations/29615557
http://purl.uniprot.org/uniprot/#_P28465-mappedCitation-29615557http://www.w3.org/1999/02/22-rdf-syntax-ns#objecthttp://purl.uniprot.org/citations/29615557