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http://purl.uniprot.org/citations/30228288http://www.w3.org/1999/02/22-rdf-syntax-ns#typehttp://purl.uniprot.org/core/Journal_Citation
http://purl.uniprot.org/citations/30228288http://www.w3.org/2000/01/rdf-schema#comment"Fibroblast growth factor 23 (FGF23) is associated with cardiovascular disease and all-cause mortality in patients with diabetes mellitus. Insulin resistance has recently been reported to increase FGF23 levels, and resistin is a peptide that mainly regulates insulin resistance. However, few studies have investigated the association between FGF23 and resistin. A total of 422 patients with diabetes mellitus were recruited for this cross-sectional study to examine the association between resistin and intact FGF23. The mean ( ± standard deviation) age was 63.1 ± 11.9 years, and the median HbA1c was 6.7% (range, 6.1-7.1%). The mean estimated glomerular filtration rate (eGFR) was 66.2 ± 23.1 mL/min/m2. Multiple regression analysis for resistin showed that logFGF23 (coefficient (Coef): 1.551; standard error (SE): 0.739; P = 0.036), C-peptide (Coef: 0.798; SE: 0.229; P = 0.001), ghrelin (Coef: 1.061; SE: 0.332; P = 0.001), intact parathyroid hormone (Coef: 0.022; SE: 0.099; P = 0.030), and eGFR (Coef: -0.091; SE: 0.017; P < 0.001) were all significantly associated with the resistin level. These associations were modified in patients with higher age, lower body mass index, and higher vitamin D levels. These results suggest that resistin is positively associated with serum FGF23 levels."xsd:string
http://purl.uniprot.org/citations/30228288http://purl.org/dc/terms/identifier"doi:10.1038/s41598-018-32432-z"xsd:string
http://purl.uniprot.org/citations/30228288http://purl.uniprot.org/core/author"Yokoyama K."xsd:string
http://purl.uniprot.org/citations/30228288http://purl.uniprot.org/core/author"Nakashima A."xsd:string
http://purl.uniprot.org/citations/30228288http://purl.uniprot.org/core/author"Yokoo T."xsd:string
http://purl.uniprot.org/citations/30228288http://purl.uniprot.org/core/author"Kawanami D."xsd:string
http://purl.uniprot.org/citations/30228288http://purl.uniprot.org/core/author"Ohkido I."xsd:string
http://purl.uniprot.org/citations/30228288http://purl.uniprot.org/core/author"Utsunomiya K."xsd:string
http://purl.uniprot.org/citations/30228288http://purl.uniprot.org/core/author"Urashima M."xsd:string
http://purl.uniprot.org/citations/30228288http://purl.uniprot.org/core/date"2018"xsd:gYear
http://purl.uniprot.org/citations/30228288http://purl.uniprot.org/core/name"Sci Rep"xsd:string
http://purl.uniprot.org/citations/30228288http://purl.uniprot.org/core/pages"13999"xsd:string
http://purl.uniprot.org/citations/30228288http://purl.uniprot.org/core/title"Association between resistin and fibroblast growth factor 23 in patients with type 2 diabetes mellitus."xsd:string
http://purl.uniprot.org/citations/30228288http://purl.uniprot.org/core/volume"8"xsd:string
http://purl.uniprot.org/citations/30228288http://www.w3.org/2004/02/skos/core#exactMatchhttp://purl.uniprot.org/pubmed/30228288
http://purl.uniprot.org/citations/30228288http://xmlns.com/foaf/0.1/primaryTopicOfhttps://pubmed.ncbi.nlm.nih.gov/30228288
http://purl.uniprot.org/uniprot/#_Q7Z4T2-mappedCitation-30228288http://www.w3.org/1999/02/22-rdf-syntax-ns#objecthttp://purl.uniprot.org/citations/30228288
http://purl.uniprot.org/uniprot/#_Q9GZV9-mappedCitation-30228288http://www.w3.org/1999/02/22-rdf-syntax-ns#objecthttp://purl.uniprot.org/citations/30228288
http://purl.uniprot.org/uniprot/#_Q9HD89-mappedCitation-30228288http://www.w3.org/1999/02/22-rdf-syntax-ns#objecthttp://purl.uniprot.org/citations/30228288
http://purl.uniprot.org/uniprot/Q9GZV9http://purl.uniprot.org/core/mappedCitationhttp://purl.uniprot.org/citations/30228288
http://purl.uniprot.org/uniprot/Q9HD89http://purl.uniprot.org/core/mappedCitationhttp://purl.uniprot.org/citations/30228288
http://purl.uniprot.org/uniprot/Q7Z4T2http://purl.uniprot.org/core/mappedCitationhttp://purl.uniprot.org/citations/30228288