| http://purl.uniprot.org/citations/8613139 | http://www.w3.org/1999/02/22-rdf-syntax-ns#type | http://purl.uniprot.org/core/Journal_Citation |
| http://purl.uniprot.org/citations/8613139 | http://www.w3.org/1999/02/22-rdf-syntax-ns#type | http://purl.uniprot.org/core/Journal_Citation |
| http://purl.uniprot.org/citations/8613139 | http://www.w3.org/2000/01/rdf-schema#comment | "The ability to develop type II collagen (CII)-induced arthritis (CIA) in mice is associated with the major histocompatibility I-A gene and with as yet poorly defined regulatory molecules of the major histocompatibility complex (MHC) class II antigen processing and presentation pathway. H2-M molecules are thought to be involved in the loading of antigenic peptides into the MHC class II binding cleft. We sequenced H2-Ma, H2-Mb1, and H2-Mb2 genes from CIA-susceptible and -resistant mouse strains and identified four different Ma and Mb2 alleles and three different Mb1 alleles defined by polymorphic residues within the predicted peptide binding groove. Most CIA-resistant mouse strains share common Ma, Mb1, and Mb2 alleles. In contrast, H2-M alleles designated Ma-III, Ma-IV, Mb1-III, and Mb2-IV could be exclusively identified in the CIA-susceptible H2r and H2q haplotypes, suggesting that allelic H2-M molecules may modulate the composition of different CII peptides loaded into MHC class II molecules, presumably presenting "arthritogenic" epitopes to T lymphocytes."xsd:string |
| http://purl.uniprot.org/citations/8613139 | http://purl.org/dc/terms/identifier | "doi:10.1007/s002510050085"xsd:string |
| http://purl.uniprot.org/citations/8613139 | http://purl.org/dc/terms/identifier | "doi:10.1007/s002510050085"xsd:string |
| http://purl.uniprot.org/citations/8613139 | http://purl.uniprot.org/core/author | "Loos M."xsd:string |
| http://purl.uniprot.org/citations/8613139 | http://purl.uniprot.org/core/author | "Loos M."xsd:string |
| http://purl.uniprot.org/citations/8613139 | http://purl.uniprot.org/core/author | "Walter W."xsd:string |
| http://purl.uniprot.org/citations/8613139 | http://purl.uniprot.org/core/author | "Walter W."xsd:string |
| http://purl.uniprot.org/citations/8613139 | http://purl.uniprot.org/core/author | "Maeurer M.J."xsd:string |
| http://purl.uniprot.org/citations/8613139 | http://purl.uniprot.org/core/author | "Maeurer M.J."xsd:string |
| http://purl.uniprot.org/citations/8613139 | http://purl.uniprot.org/core/date | "1996"xsd:gYear |
| http://purl.uniprot.org/citations/8613139 | http://purl.uniprot.org/core/date | "1996"xsd:gYear |
| http://purl.uniprot.org/citations/8613139 | http://purl.uniprot.org/core/name | "Immunogenetics"xsd:string |
| http://purl.uniprot.org/citations/8613139 | http://purl.uniprot.org/core/name | "Immunogenetics"xsd:string |
| http://purl.uniprot.org/citations/8613139 | http://purl.uniprot.org/core/pages | "19-26"xsd:string |
| http://purl.uniprot.org/citations/8613139 | http://purl.uniprot.org/core/pages | "19-26"xsd:string |
| http://purl.uniprot.org/citations/8613139 | http://purl.uniprot.org/core/title | "H2-M polymorphism in mice susceptible to collagen-induced arthritis involves the peptide binding groove."xsd:string |
| http://purl.uniprot.org/citations/8613139 | http://purl.uniprot.org/core/title | "H2-M polymorphism in mice susceptible to collagen-induced arthritis involves the peptide binding groove."xsd:string |
| http://purl.uniprot.org/citations/8613139 | http://purl.uniprot.org/core/volume | "44"xsd:string |
| http://purl.uniprot.org/citations/8613139 | http://purl.uniprot.org/core/volume | "44"xsd:string |
| http://purl.uniprot.org/citations/8613139 | http://www.w3.org/2004/02/skos/core#exactMatch | http://purl.uniprot.org/pubmed/8613139 |
| http://purl.uniprot.org/citations/8613139 | http://www.w3.org/2004/02/skos/core#exactMatch | http://purl.uniprot.org/pubmed/8613139 |
| http://purl.uniprot.org/citations/8613139 | http://xmlns.com/foaf/0.1/primaryTopicOf | https://pubmed.ncbi.nlm.nih.gov/8613139 |
| http://purl.uniprot.org/citations/8613139 | http://xmlns.com/foaf/0.1/primaryTopicOf | https://pubmed.ncbi.nlm.nih.gov/8613139 |