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http://purl.uniprot.org/citations/9052782http://www.w3.org/1999/02/22-rdf-syntax-ns#typehttp://purl.uniprot.org/core/Journal_Citation
http://purl.uniprot.org/citations/9052782http://www.w3.org/1999/02/22-rdf-syntax-ns#typehttp://purl.uniprot.org/core/Journal_Citation
http://purl.uniprot.org/citations/9052782http://www.w3.org/2000/01/rdf-schema#comment"Endothelial dysfunction or activation elicited by oxidatively modified low-density lipoprotein (Ox-LDL) has been implicated in the pathogenesis of atherosclerosis, characterized by intimal thickening and lipid deposition in the arteries. Ox-LDL and its lipid constituents impair endothelial production of nitric oxide, and induce the endothelial expression of leukocyte adhesion molecules and smooth-muscle growth factors, which may be involved in atherogenesis. Vascular endothelial cells in culture and in vivo internalize and degrade Ox-LDL through a putative receptor-mediated pathway that does not involve macrophage scavenger receptors. Here we report the molecular cloning, using expression cloning strategy, of an Ox-LDL receptor from vascular endothelial cells. The cloned receptor is a membrane protein that belongs structurally to the C-type lectin family, and is expressed in vivo in vascular endothelium and vascular-rich organs."xsd:string
http://purl.uniprot.org/citations/9052782http://purl.org/dc/terms/identifier"doi:10.1038/386073a0"xsd:string
http://purl.uniprot.org/citations/9052782http://purl.org/dc/terms/identifier"doi:10.1038/386073a0"xsd:string
http://purl.uniprot.org/citations/9052782http://purl.uniprot.org/core/author"Masaki T."xsd:string
http://purl.uniprot.org/citations/9052782http://purl.uniprot.org/core/author"Masaki T."xsd:string
http://purl.uniprot.org/citations/9052782http://purl.uniprot.org/core/author"Tanaka T."xsd:string
http://purl.uniprot.org/citations/9052782http://purl.uniprot.org/core/author"Tanaka T."xsd:string
http://purl.uniprot.org/citations/9052782http://purl.uniprot.org/core/author"Miwa S."xsd:string
http://purl.uniprot.org/citations/9052782http://purl.uniprot.org/core/author"Miwa S."xsd:string
http://purl.uniprot.org/citations/9052782http://purl.uniprot.org/core/author"Kita T."xsd:string
http://purl.uniprot.org/citations/9052782http://purl.uniprot.org/core/author"Kita T."xsd:string
http://purl.uniprot.org/citations/9052782http://purl.uniprot.org/core/author"Aoyama T."xsd:string
http://purl.uniprot.org/citations/9052782http://purl.uniprot.org/core/author"Aoyama T."xsd:string
http://purl.uniprot.org/citations/9052782http://purl.uniprot.org/core/author"Katsura Y."xsd:string
http://purl.uniprot.org/citations/9052782http://purl.uniprot.org/core/author"Katsura Y."xsd:string
http://purl.uniprot.org/citations/9052782http://purl.uniprot.org/core/author"Aiba Y."xsd:string
http://purl.uniprot.org/citations/9052782http://purl.uniprot.org/core/author"Aiba Y."xsd:string
http://purl.uniprot.org/citations/9052782http://purl.uniprot.org/core/author"Sawamura T."xsd:string
http://purl.uniprot.org/citations/9052782http://purl.uniprot.org/core/author"Sawamura T."xsd:string
http://purl.uniprot.org/citations/9052782http://purl.uniprot.org/core/author"Kume N."xsd:string
http://purl.uniprot.org/citations/9052782http://purl.uniprot.org/core/author"Kume N."xsd:string
http://purl.uniprot.org/citations/9052782http://purl.uniprot.org/core/author"Moriwaki H."xsd:string
http://purl.uniprot.org/citations/9052782http://purl.uniprot.org/core/author"Moriwaki H."xsd:string