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http://purl.uniprot.org/citations/9389651http://www.w3.org/1999/02/22-rdf-syntax-ns#typehttp://purl.uniprot.org/core/Journal_Citation
http://purl.uniprot.org/citations/9389651http://www.w3.org/1999/02/22-rdf-syntax-ns#typehttp://purl.uniprot.org/core/Journal_Citation
http://purl.uniprot.org/citations/9389651http://www.w3.org/2000/01/rdf-schema#comment"The Arabidopsis gai mutant allele confers a reduction in gibberellin (GA) responsiveness. Here we report the molecular cloning of GAI and a closely related gene GRS. The predicted GAI (wild-type) and gai (mutant) proteins differ only by the deletion of a 17-amino-acid segment from within the amino-terminal region. GAI and GRS contain nuclear localization signals, a region of homology to a putative transcription factor, and motifs characteristic of transcriptional coactivators. Genetic analysis indicates that GAI is a repressor of GA responses, that GA can release this repression, and that gai is a mutant repressor that is relatively resistant to the effects of GA. Mutations at SPY and GAR2 suppress the gai phenotype, indicating the involvement of GAI, SPY, and GAR2 in a signaling pathway that regulates GA responses negatively. The existence of this pathway suggests that GA modulates plant growth through derepression rather than through simple stimulation."xsd:string
http://purl.uniprot.org/citations/9389651http://purl.org/dc/terms/identifier"doi:10.1101/gad.11.23.3194"xsd:string
http://purl.uniprot.org/citations/9389651http://purl.org/dc/terms/identifier"doi:10.1101/gad.11.23.3194"xsd:string
http://purl.uniprot.org/citations/9389651http://purl.uniprot.org/core/author"Murphy G.P."xsd:string
http://purl.uniprot.org/citations/9389651http://purl.uniprot.org/core/author"Murphy G.P."xsd:string
http://purl.uniprot.org/citations/9389651http://purl.uniprot.org/core/author"Peng J."xsd:string
http://purl.uniprot.org/citations/9389651http://purl.uniprot.org/core/author"Peng J."xsd:string
http://purl.uniprot.org/citations/9389651http://purl.uniprot.org/core/author"Carol P."xsd:string
http://purl.uniprot.org/citations/9389651http://purl.uniprot.org/core/author"Carol P."xsd:string
http://purl.uniprot.org/citations/9389651http://purl.uniprot.org/core/author"Harberd N.P."xsd:string
http://purl.uniprot.org/citations/9389651http://purl.uniprot.org/core/author"Harberd N.P."xsd:string
http://purl.uniprot.org/citations/9389651http://purl.uniprot.org/core/author"Richards D.E."xsd:string
http://purl.uniprot.org/citations/9389651http://purl.uniprot.org/core/author"Richards D.E."xsd:string
http://purl.uniprot.org/citations/9389651http://purl.uniprot.org/core/author"Cowling R.J."xsd:string
http://purl.uniprot.org/citations/9389651http://purl.uniprot.org/core/author"Cowling R.J."xsd:string
http://purl.uniprot.org/citations/9389651http://purl.uniprot.org/core/author"King K.E."xsd:string
http://purl.uniprot.org/citations/9389651http://purl.uniprot.org/core/author"King K.E."xsd:string
http://purl.uniprot.org/citations/9389651http://purl.uniprot.org/core/date"1997"xsd:gYear
http://purl.uniprot.org/citations/9389651http://purl.uniprot.org/core/date"1997"xsd:gYear
http://purl.uniprot.org/citations/9389651http://purl.uniprot.org/core/name"Genes Dev."xsd:string
http://purl.uniprot.org/citations/9389651http://purl.uniprot.org/core/name"Genes Dev."xsd:string
http://purl.uniprot.org/citations/9389651http://purl.uniprot.org/core/pages"3194-3205"xsd:string
http://purl.uniprot.org/citations/9389651http://purl.uniprot.org/core/pages"3194-3205"xsd:string