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| http://purl.uniprot.org/citations/9469811 | http://www.w3.org/2000/01/rdf-schema#comment | "Mice homozygous for a disrupted allele of the mismatch repair gene Pms2 have a mutator phenotype. When this allele is crossed into quasi-monoclonal (QM) mice, which have a very limited B cell repertoire, homozygotes have fewer somatic mutations at the immunoglobulin heavy chain and lambda chain loci than do heterozygotes or wild-type QM mice. That is, mismatch repair seems to contribute to somatic hypermutation rather than stifling it. It is suggested that at immunoglobulin loci in hypermutable B cells, mismatched base pairs are "corrected" according to the newly synthesized DNA strand, thereby fixing incipient mutations instead of eliminating them."xsd:string |
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| http://purl.uniprot.org/citations/9469811 | http://purl.uniprot.org/core/author | "Wong J."xsd:string |
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| http://purl.uniprot.org/citations/9469811 | http://purl.uniprot.org/core/date | "1998"xsd:gYear |
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